Overexpression of Nitric Oxide Synthase Restores Circulating Angiogenic Cell Function in Patients With Coronary Artery Disease: Implications for Autologous Cell Therapy for Myocardial Infarction Article

Chen, Qiumei, Varga, Monika, Wang, Xiaoyin et al. (2016). Overexpression of Nitric Oxide Synthase Restores Circulating Angiogenic Cell Function in Patients With Coronary Artery Disease: Implications for Autologous Cell Therapy for Myocardial Infarction . 5(1), 10.1161/JAHA.115.002257

Open Access International Collaboration

cited authors

  • Chen, Qiumei; Varga, Monika; Wang, Xiaoyin; Haddad, Daniel J; An, Songtao; Medzikovic, Lejla; Derakhshandeh, Ronak; Kostyushev, Dmitry S; Zhang, Yan; Clifford, Brian T; Luu, Emmy; Danforth, Olivia M; Rafikov, Ruslan; Gong, Wenhui; Black, Stephen M; Suchkov, Sergey V; Fineman, Jeffrey R; Heiss, Christian; Aschbacher, Kirstin; Yeghiazarians, Yerem; Springer, Matthew L

fiu authors

publication date

  • January 1, 2016

keywords

  • BONE-MARROW
  • CAPACITY
  • Cardiac & Cardiovascular Systems
  • Cardiovascular System & Cardiology
  • ENDOTHELIAL PROGENITOR CELLS
  • GENE-TRANSFER
  • Life Sciences & Biomedicine
  • MARROW MONONUCLEAR-CELLS
  • MOBILIZATION
  • NEOVASCULARIZATION
  • NO SYNTHASE
  • Science & Technology
  • TOPCARE-AMI
  • TRANSPLANTATION
  • circulating angiogenic cells
  • endothelial progenitor cells
  • gene therapy
  • myocardial infarction
  • nitric oxide synthase

Digital Object Identifier (DOI)

volume

  • 5

issue

  • 1